New Research on Vitamin D, Part 1: Vitamin D is Unique | WholeFoods Magazine

New Research on Vitamin D, Part 1: Vitamin D is Unique | WholeFoods Magazine
The vitamin D precursor in our skin is 7-dehydrocholesterol, which is made in the body from cholesterol. Ultraviolet-B (UVB) energy converts 7-dehydrocholesterol into cholecalciferol, which is the name of natural vitamin D. Cholecalciferol (pronounced koh· luh· kal· sif· uh· rawl) is then carried in the bloodstream to the liver, where it is converted into the prohormone 25-hydroxy vitamin D (calcidiol). Some of that 25-hydroxy vitamin D is then transported to the kidneys where it is hydroxylated further to form 1,25-hydroxy vitamin D (calcitriol). This form is the physiologically active form of vitamin D. The calcitriol made in the kidneys is to fulfill only one of vitamin D’s functions: its endocrine function, which is to maintain the calcium economy.

Technically not a “vitamin,” vitamin D is in a class by itself. Its second metabolic product, 1,25-hydroxy vitamin D (calcitriol), is actually a hormone, a key that fits binding sites on the human genome. The human genome contains more than 2,700 binding sites for 1,25-hydroxy vitamin D; those binding sites are near genes involved in virtually every known major disease of humans.

Ryan McLaughlin's Shine on Scotland campaign

TEENAGER OF COURAGE

He spearheaded a campaign to educate people on the importance of Vitamin D in combating MS.

The Pride of Britain Awards - Ryan McLaughlin

Vitamin D 'triggers and arms' the immune system

NEW RESEARCH...Vitamin D is crucial to the fending off of infections! Simply amazing.
Scientists at the University of Copenhagen have discovered that Vitamin D is crucial to activating our immune defences and that without sufficient intake of the vitamin, the killer cells of the immune system – T cells – will not be able to react to and fight off serious infections in the body.

For T cells to detect and kill foreign pathogens such as clumps of bacteria or viruses, the cells must first be ‘triggered’ into action and "transform" from inactive and harmless immune cells into killer cells that are primed to seek out and destroy all traces of invaders.

The researchers found that the T cells rely on vitamin D in order activate and they would remain dormant, ‘naïve’ to the possibility of threat if vitamin D is lacking in the blood.

Professor Carsten Geisler from the Department of International Health, Immunology and Microbiology, said: "When a T cell is exposed to a foreign pathogen, it extends a signalling device or ‘antenna’ known as a vitamin D receptor, with which it searches for vitamin D.

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